Academic Appointments

Administrative Appointments

  • Consulting Professor, Stanford Dept. Health Policy & Research (2007 - Present)
  • Senior Research Scientist, Cancer Prevention Institute of California (2006 - Present)

Professional Education

  • Ph.D., University of California, Berkeley, Epidemiology (1983)
  • MPH, University of California, Berkeley, Behavioral Science (1978)
  • BA, University of California, Santa Cruz, Psychology (1968)

Research & Scholarship

Current Research and Scholarly Interests

Dr. Reynolds directs the environmental research group at CPIC, with primary research interests in the environmental influences in the etiology of breast cancer and cancers in children. She has also conducted research in the role of second hand smoking in the development of cancers of the lung, breast and colon.


All Publications

  • Birth Weight and Order as Risk Factors for Childhood Central Nervous System Tumors JOURNAL OF PEDIATRICS Maclean, J., Partap, S., Reynolds, P., Von Behren, J., Fisher, P. G. 2010; 157 (3): 450-455


    To determine whether birth characteristics related to maternal-fetal health in utero are associated with the development of childhood central nervous system tumors.We identified, from the California Cancer Registry, 3733 children under age 15 diagnosed with childhood central nervous system tumors between 1988 and 2006 and linked these cases to their California birth certificates. Four controls per case, matched on birth date and sex, were randomly selected from the same birth files. We evaluated associations of multiple childhood CNS tumor subtypes with birth weight and birth order.Low birth weight was associated with a reduced risk of low-grade gliomas (OR=0.67; 95% CI, 0.46 to 0.97) and high birth weight was associated with increased risk of high-grade gliomas (OR=1.57; 95% CI, 1.16 to 2.12). High birth order (fourth or higher) was associated with decreased risk of low-grade gliomas (OR=0.75; 95% CI, 0.56 to 0.99) and increased risk of high-grade gliomas (OR=1.32; 95% CI, 1.01 to 1.72 for second order).Factors that drive growth in utero may increase the risk of low-grade gliomas. There may be a similar relationship in high-grade gliomas, although other factors, such as early infection, may modify this association. Additional investigation is warranted to validate and further define these findings.

    View details for DOI 10.1016/j.jpeds.2010.04.006

    View details for Web of Science ID 000281116100023

    View details for PubMedID 20553692

  • Residential proximity to agricultural pesticide applications and childhood acute lymphoblastic leukemia ENVIRONMENTAL RESEARCH Rull, R. P., Gunier, R., Von Behren, J., Hertz, A., Crouse, V., Buffler, P. A., Reynolds, P. 2009; 109 (7): 891-899


    Ambient exposure from residential proximity to applications of agricultural pesticides may contribute to the risk of childhood acute lymphoblastic leukemia (ALL). Using residential histories collected from the families of 213 ALL cases and 268 matched controls enrolled in the Northern California Childhood Leukemia Study, the authors assessed residential proximity within a half-mile (804.5m) of pesticide applications by linking address histories with reports of agricultural pesticide use. Proximity was ascertained during different time windows of exposure, including the first year of life and the child's lifetime through the date of diagnosis for cases or reference for controls. Agricultural pesticides were categorized a priori into groups based on similarities in toxicological effects, physicochemical properties, and target pests or uses. The effects of moderate and high exposure for each group of pesticides were estimated using conditional logistic regression. Elevated ALL risk was associated with lifetime moderate exposure, but not high exposure, to certain physicochemical categories of pesticides, including organophosphates, chlorinated phenols, and triazines, and with pesticides classified as insecticides or fumigants. A similar pattern was also observed for several toxicological groups of pesticides. These findings suggest future directions for the identification of specific pesticides that may play a role in the etiology of childhood leukemia.

    View details for DOI 10.1016/j.envres.2009.07.014

    View details for Web of Science ID 000270252600014

    View details for PubMedID 19700145

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